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The influence of loss of interruption variants on astrocyte reactivity in Huntington disease

The Huntington disease (HD) mutation produces a pathogenic mutant HTT (mHTT) protein with an elongated polyglutamine tract. Presence of mHTT disrupts diverse cellular pathways, leading to adverse molecular phenotypes including inflammation, impaired synaptic transmission, and eventually neurodegeneration. Although HD has a singular genetic cause, there have been recent advances in our understanding of HD gene modifiers (PMID: 26232222).
Projet en cours Voir le projet et les chercheurs
TYPE DE SUBVENTION
Subventions pour le renforcement des capacités
Domaine de recherche
Neurodégénérescence
Concours
2026 Huntington Society of Canada - Brain Canada Undergraduate Student Summer Fellowship
Province
Colombie-Britannique
DATE DE DÉBUT
2026