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The influence of loss of interruption variants on astrocyte reactivity in Huntington disease

The Huntington disease (HD) mutation produces a pathogenic mutant HTT (mHTT) protein with an elongated polyglutamine tract. Presence of mHTT disrupts diverse cellular pathways, leading to adverse molecular phenotypes including inflammation, impaired synaptic transmission, and eventually neurodegeneration. Although HD has a singular genetic cause, there have been recent advances in our understanding of HD gene modifiers (PMID: 26232222).
Project ongoing See Project & Researchers
Grant type
Capacity building grants
Area of Research
Neurodegeneration
Competition
2026 Huntington Society of Canada - Brain Canada Undergraduate Student Summer Fellowship
Province
British Columbia
Start Date
2026